The P1D1 Gene In Relation To Brain Cancer

By Rob Sutter


When talking about brain cancer on the whole, the P1D1 gene is one idea that is more than worth considering. Typically, it is put to use in order to send insulin throughout the body while regulating it, in addition to providing mitochondrial function. This gene has been focused on in studies before but it seems like they have been more rooted in ideas like obesity and diabetes more so than anything else. Can the same be said about this particular type of cancer as well?

It's not hard to see why this gene could potentially become linked to brain cancer, as you look at the various stories on the matter. Therapies have been constructed in order to help those with tumors and it seems like each of them have been able to possess certain levels of success. In fact, said therapies are ones which organizations like Voices against Brain Cancer can bring to your attention. With that said, what makes the P1D1 so integral when it comes to helping those with cancer?

There was a report posted on Business Wire and it spoke, in detail, about the level of messenger RNA that can be found in the gene referred to as P1D1. For those who do not know, this can play greatly in terms of two conditions in particular: medulloblastomas and gliomas. P1D1 is needed in order to inhibit the growth of cells in said conditions, which speaks volumes about their functionality. With a study set in place, what can be said about this gene in relation to cancer in general?

With the results of the therapy brought to the forefront, greater levels of P1D1 mRNA have been connected to the increases in survival rates of patients. The way that P1D1 works is when seen in high doses can actually work in order to kill tumor cells and stop the proliferation of the cells as well. With many adults studied, according to the article, each of them was able to see better survival rates than they would have had otherwise. It is clear that the presence of this gene is an idea that calls for attention.

These types of therapies are done consistently and I do not think that anyone can speak against the levels of potential that they possess. To me, they are able to address this condition, which has normally been viewed as one of the most serious in the human body. While conventional surgery on its own exists, results are most likely going to vary when looking at a collective group of patients. Perhaps this is one of the reasons why looking more into the P1D1 gene is for the best.




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